Abstract
Burn injury is accompanied by complex pathophysiological alterations that exert deleterious effects on various organ systems (1). Inflammatory shock mediators that are implicated in the pathogenesis of burn shock include histamine, serotonin, kinins, oxygen free radicals (OFR), prostaglandins, thromboxane and interleukins (2).
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This is an open access article distributed under the Creative Commons Attribution License which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Article Type: Case Report
EUR J GEN MED, 2004, Volume 1, Issue 1, 26-27
https://doi.org/10.29333/ejgm/82162
Publication date: 15 Jan 2004
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